TY  - JOUR 
AU  - Rodriguez-Sevilla, Juan Jose
AU  - Ganan-Gomez, Irene
AU  - Ma, Feiyang
AU  - Chien, Kelly
AU  - Del Rey,  Monica
AU  - Loghavi, Sanam
AU  - Montalban-Bravo, Guillermo
AU  - Adema, Vera
AU  - Wildeman, Bethany
AU  - Kanagal-Shamanna, Rashmi
AU  - Bazinet, Alexandre
AU  - Chifotides, Helen T
AU  - Thongon, Natthakan
AU  - Calvo, Xavier
AU  - Hernández Rivas, Jesús María
AU  - Díez Campelo, María
AU  - Garcia-Manero, Guillermo
AU  - Colla, Simona
PY  - 2024
UR  - http://hdl.handle.net/10366/172058
AB  - [EN]The molecular mechanisms of venetoclax-based therapy failure in patients with acute myeloid leukemia were recently clarified, but the mechanisms by which patients with myelodysplastic syndromes (MDS) acquire secondary resistance to venetoclax...
LA  - eng
KW  - Myelodysplastic Syndromes
KW  - Leukemia, Myeloid, Acute
KW  - Humans
KW  - Hematopoietic Stem Cells
KW  - Bridged Bicyclo Compounds, Heterocyclic
KW  - Sulfonamides
KW  - Sulfonamides
KW  - Heterocyclic Compounds
KW  - Leukemia
KW  - Humans
KW  - Myelodysplastic Syndromes
KW  - Hematopoietic Stem Cells
TI  - Hematopoietic stem cells with granulo-monocytic differentiation state overcome venetoclax sensitivity in patients with myelodysplastic syndromes
DO  - 10.1038/s41467-024-46424-3
T2  - Nature communications
VL  - 15
M2  - 2428
ER  -