TY  - JOUR 
AU  - García Domingo, Mónica
AU  - Morán Benito, Asunción
AU  - Martín Calvo, María Luisa
AU  - Barthelmebs, Mariette
AU  - San Román, Luis
PY  - 2006
SN  - 0014-2999
UR  - http://hdl.handle.net/10366/159278
AB  - [EN]We investigated the involvement of the nitric oxide pathway in the inhibitory mechanisms of 5-hydroxytryptamine (5-HT) in the pressor responses induced by stimulation of sympathetic vasopressor outflow in diabetic pithed rats. Diabetes was induced...
LA  - eng
KW  - 5-hydroxytryptamine
KW  - Nitric oxide
KW  - Experimental diabetes
KW  - 5-HT1A receptors
KW  - Prejunctional inhibition
KW  - NG-Nitroarginine Methyl Ester
KW  - Nitric Oxide Synthase
KW  - Diabetes Mellitus
KW  - Electric Stimulation
KW  - 8-Hydroxy-2-(di-n-propylamino)tetralin
KW  - Atropine
KW  - Norepinephrine
KW  - Nitric Oxide
KW  - Blood Pressure
KW  - Oxadiazoles
KW  - Serotonin
KW  - Enzyme Inhibitors
KW  - Quinoxalines
KW  - Spinal Cord
KW  - Muscarinic Antagonists
KW  - Guanylate Cyclase
KW  - Rats
KW  - Animals
KW  - Serotonin 5-HT1 Receptor Agonists
KW  - Vasoconstriction
KW  - Tryptamines
KW  - Serotonin Receptor Agonists
TI  - The nitric oxide synthesis/pathway mediates the inhibitory serotoninergic responses of the pressor effect elicited by sympathetic stimulation in diabetic pithed rats.
DO  - 10.1016/j.ejphar.2006.03.020
T2  - European Journal of Pharmacology
VL  - 537
M2  - 126
ER  -